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Trp53 Haploinsufficiency Modifies EGFR-Driven Peripheral Nerve Sheath Tumorigenesis
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Conditional Inactivation of Pten with EGFR Overexpression in Schwann Cells Models Sporadic MPNST.
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Simple and efficient methods for enrichment and isolation of endonuclease modified cells
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Genetically engineered minipigs model the major clinical features of human neurofibromatosis type 1
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Identification of candidate neoantigens produced by fusion transcripts in human osteosarcomas
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David Alexander Lubbock
Charlotte Lubbock
Harriet Victoria Lubbock
Richard Watson
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Forward genetic screen for malignant peripheral nerve sheath tumor formation identifies new genes and pathways driving tumorigenesis
Canonical Wnt/β-catenin signaling drives human schwann cell transformation, progression, and tumor maintenance
Trp53 Haploinsufficiency Modifies EGFR-Driven Peripheral Nerve Sheath Tumorigenesis
Conditional Inactivation of Pten with EGFR Overexpression in Schwann Cells Models Sporadic MPNST.
Simple and efficient methods for enrichment and isolation of endonuclease modified cells
Genetically engineered minipigs model the major clinical features of human neurofibromatosis type 1
Identification of candidate neoantigens produced by fusion transcripts in human osteosarcomas
MMuFLR: missense mutation and frameshift location reporter